Exogenous S1P Exposure Potentiates Ischemic Stroke Damage That Is Reduced Possibly by Inhibiting S1P Receptor Signaling

Initial and recurrent stroke produces central nervous system (CNS) damage, involving neuroinflammation. Receptor-mediated S1P signaling can influence neuroinflammation and has been implicated in cerebral ischemia through effects on the immune system. However, S1P-mediated events also occur within th...

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Detalles Bibliográficos
Autores principales: Moon, Eunjung, Han, Jeong Eun, Jeon, Sejin, Ryu, Jong Hoon, Choi, Ji Woong, Chun, Jerold
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Hindawi Publishing Corporation 2015
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4630407/
https://www.ncbi.nlm.nih.gov/pubmed/26576074
http://dx.doi.org/10.1155/2015/492659

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4630407/
https://www.ncbi.nlm.nih.gov/pubmed/26576074
http://dx.doi.org/10.1155/2015/492659

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