MARCKS Signaling Differentially Regulates Vascular Smooth Muscle and Endothelial Cell Proliferation through a KIS-, p27(kip1)- Dependent Mechanism

BACKGROUND: Overexpression of the myristolated alanine-rich C kinase substrate (MARCKS) occurs in vascular proliferative diseases such as restenosis after bypass surgery. MARCKS knockdown results in arrest of vascular smooth muscle cell (VSMC) proliferation with little effect on endothelial cell (EC...

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Detalles Bibliográficos
Autores principales: Yu, Dan, Makkar, George, Dong, Tuo, Strickland, Dudley K., Sarkar, Rajabrata, Monahan, Thomas Stacey
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Public Library of Science 2015
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4631550/
https://www.ncbi.nlm.nih.gov/pubmed/26528715
http://dx.doi.org/10.1371/journal.pone.0141397

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4631550/
https://www.ncbi.nlm.nih.gov/pubmed/26528715
http://dx.doi.org/10.1371/journal.pone.0141397

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