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MARCKS Signaling Differentially Regulates Vascular Smooth Muscle and Endothelial Cell Proliferation through a KIS-, p27(kip1)- Dependent Mechanism
BACKGROUND: Overexpression of the myristolated alanine-rich C kinase substrate (MARCKS) occurs in vascular proliferative diseases such as restenosis after bypass surgery. MARCKS knockdown results in arrest of vascular smooth muscle cell (VSMC) proliferation with little effect on endothelial cell (EC...
Autores principales: | Yu, Dan, Makkar, George, Dong, Tuo, Strickland, Dudley K., Sarkar, Rajabrata, Monahan, Thomas Stacey |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2015
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4631550/ https://www.ncbi.nlm.nih.gov/pubmed/26528715 http://dx.doi.org/10.1371/journal.pone.0141397 |
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