Cellular oxidative stress response mediates radiosensitivity in Fus1-deficient mice

Mechanism of radiosensitivity of normal tissues, a key factor in determining the toxic side effects of cancer radiotherapy, is not fully understood. We recently demonstrated that deficiency of mitochondrial tumor suppressor, Fus1, increases radiosensitivity at the organismal, tissue and cellular lev...

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Detalles Bibliográficos
Autores principales: Yazlovitskaya, E M, Voziyan, P A, Manavalan, T, Yarbrough, W G, Ivanova, A V
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group 2015
Materias:
Original Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4669799/
https://www.ncbi.nlm.nih.gov/pubmed/25695605
http://dx.doi.org/10.1038/cddis.2014.593

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4669799/
https://www.ncbi.nlm.nih.gov/pubmed/25695605
http://dx.doi.org/10.1038/cddis.2014.593

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