Overexpression of myeloid differentiation protein 88 in mice induces mild cardiac dysfunction, but no deficit in heart morphology

Cardiac remodeling involves changes in heart shape, size, structure, and function after injury to the myocardium. The proinflammatory adaptor protein myeloid differentiation protein 88 (MyD88) contributes to cardiac remodeling. To investigate whether excessive MyD88 levels initiate spontaneous cardi...

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Detalles Bibliográficos
Autores principales: Chen, W., Huang, Z., Jiang, X., Li, C., Gao, X.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Associação Brasileira de Divulgação Científica 2015
Materias:
Biomedical Sciences
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4681416/
https://www.ncbi.nlm.nih.gov/pubmed/26628395
http://dx.doi.org/10.1590/1414-431X20154794

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4681416/
https://www.ncbi.nlm.nih.gov/pubmed/26628395
http://dx.doi.org/10.1590/1414-431X20154794

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