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Activation of γ-aminobutyric Acid (A) Receptor Protects Hippocampus from Intense Exercise-induced Synapses Damage and Apoptosis in Rats

BACKGROUND: Our previous study has confirmed that one bout of exhaustion (Ex) can cause hippocampus neurocyte damage, excessive apoptosis, and dysfunction. Its initial reason is intracellular calcium overload in hippocampus triggered by N-methyl-D-aspartic acid receptor (NMDAR) over-activation. NMDA...

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Detalles Bibliográficos
Autores principales: Ding, Yi, Xie, Lan, Chang, Cun-Qing, Chen, Zhi-Min, Ai, Hua
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Medknow Publications & Media Pvt Ltd 2015
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4733790/
https://www.ncbi.nlm.nih.gov/pubmed/26315081
http://dx.doi.org/10.4103/0366-6999.163392