Status Epilepticus Enhances Depotentiation after Fully Established LTP in an NMDAR-Dependent but GluN2B-Independent Manner

N-Methyl-D-aspartate (NMDA) receptor-dependent long-term potentiation (LTP) can be reversed by low-frequency stimulation (LFS) referred to as depotentiation (DP). We previously found GluN2B upregulated in CA1 neurons from post-status epilepticus (post-SE) tissue associated with an enhanced LTP. Here...

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Detalles Bibliográficos
Autores principales: Guli, Xiati, Tokay, Tursonjan, Kirschstein, Timo, Köhling, Rüdiger
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Hindawi Publishing Corporation 2016
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4735914/
https://www.ncbi.nlm.nih.gov/pubmed/26881126
http://dx.doi.org/10.1155/2016/6592038

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4735914/
https://www.ncbi.nlm.nih.gov/pubmed/26881126
http://dx.doi.org/10.1155/2016/6592038

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