Glioblastomas with copy number gains in EGFR and RNF139 show increased expressions of carbonic anhydrase genes transformed by ENO1

BACKGROUND: Prominence of glycolysis in glioblastomas may be non-specific or a feature of oncogene-related subgroups (i.e. amplified EGFR, etc.). Relationships between amplified oncogenes and expressions of metabolic genes associated with glycolysis, directly or indirectly via pH, were therefore inv...

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Detalles Bibliográficos
Autores principales: Beckner, Marie E., Pollack, Ian F., Nordberg, Mary L., Hamilton, Ronald L.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2015
Materias:
Regular Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4802406/
https://www.ncbi.nlm.nih.gov/pubmed/27051584
http://dx.doi.org/10.1016/j.bbacli.2015.11.001

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4802406/
https://www.ncbi.nlm.nih.gov/pubmed/27051584
http://dx.doi.org/10.1016/j.bbacli.2015.11.001

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