Deletion of Apoptosis Inhibitor of Macrophage (AIM)/CD5L Attenuates the Inflammatory Response and Infarct Size in Acute Myocardial Infarction

BACKGROUND: An excessive inflammatory response after myocardial infarction (MI) increases myocardial injury. The toll‐like receptor (TLR)‐4 is activated by the recognition of endogenous ligands and is proinflammatory when there is myocardial tissue injury. The apoptosis inhibitor of the macrophage (...

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Detalles Bibliográficos
Autores principales: Nishikido, Toshiyuki, Oyama, Jun‐ichi, Shiraki, Aya, Komoda, Hiroshi, Node, Koichi
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2016
Materias:
Original Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4859278/
https://www.ncbi.nlm.nih.gov/pubmed/27045005
http://dx.doi.org/10.1161/JAHA.115.002863

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4859278/
https://www.ncbi.nlm.nih.gov/pubmed/27045005
http://dx.doi.org/10.1161/JAHA.115.002863

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