TLR signaling adaptor protein MyD88 in primary sensory neurons contributes to persistent inflammatory and neuropathic pain and neuroinflammation

Increasing evidence suggests that neuro-immune and neuro-glial interactions are critically involved in chronic pain sensitization. It is well studied how immune/glial mediators sensitize pain, but how sensory neurons control neuroinflammation remains unclear. We employed Myd88 conditional knockout (...

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Detalles Bibliográficos
Autores principales: Liu, Xing-Jun, Liu, Tong, Chen, Gang, Wang, Bing, Yu, Xiao-Lu, Yin, Cui, Ji, Ru-Rong
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group 2016
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4911580/
https://www.ncbi.nlm.nih.gov/pubmed/27312666
http://dx.doi.org/10.1038/srep28188

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4911580/
https://www.ncbi.nlm.nih.gov/pubmed/27312666
http://dx.doi.org/10.1038/srep28188

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