Defective DNA repair and chromatin organization in patients with quiescent systemic lupus erythematosus

BACKGROUND: Excessive autoantibody production characterizing systemic lupus erythematosus (SLE) occurs irrespective of the disease’s clinical status and is linked to increased lymphocyte apoptosis. Herein, we tested the hypothesis that defective DNA damage repair contributes to increased apoptosis i...

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Detalles Bibliográficos
Autores principales: Souliotis, Vassilis L., Vougas, Konstantinos, Gorgoulis, Vassilis G., Sfikakis, Petros P.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2016
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4973109/
https://www.ncbi.nlm.nih.gov/pubmed/27492607
http://dx.doi.org/10.1186/s13075-016-1081-3

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4973109/
https://www.ncbi.nlm.nih.gov/pubmed/27492607
http://dx.doi.org/10.1186/s13075-016-1081-3

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