Reactive oxygen species facilitate the EDH response in arterioles by potentiating intracellular endothelial Ca(2+) release

There is abundant evidence that H(2)O(2) can act as an endothelium-derived hyperpolarizing factor in the resistance vasculature. However, whilst scavenging H(2)O(2) can abolish endothelial dependent hyperpolarization (EDH) and the associated vascular relaxation in some arteries, EDH-dependent vasore...

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Detalles Bibliográficos
Autores principales: Chidgey, James, Fraser, Paul A., Aaronson, Philip I.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier Science 2016
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5005039/
https://www.ncbi.nlm.nih.gov/pubmed/27320188
http://dx.doi.org/10.1016/j.freeradbiomed.2016.06.010

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5005039/
https://www.ncbi.nlm.nih.gov/pubmed/27320188
http://dx.doi.org/10.1016/j.freeradbiomed.2016.06.010

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