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Direct interrogation of the role of H3K9 in metazoan heterochromatin function

A defining feature of heterochromatin is methylation of Lys9 of histone H3 (H3K9me), a binding site for heterochromatin protein 1 (HP1). Although H3K9 methyltransferases and HP1 are necessary for proper heterochromatin structure, the specific contribution of H3K9 to heterochromatin function and anim...

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Autores principales: Penke, Taylor J.R., McKay, Daniel J., Strahl, Brian D., Matera, A. Gregory, Duronio, Robert J.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Cold Spring Harbor Laboratory Press 2016
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5024684/
https://www.ncbi.nlm.nih.gov/pubmed/27566777
http://dx.doi.org/10.1101/gad.286278.116
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author Penke, Taylor J.R.
McKay, Daniel J.
Strahl, Brian D.
Matera, A. Gregory
Duronio, Robert J.
author_facet Penke, Taylor J.R.
McKay, Daniel J.
Strahl, Brian D.
Matera, A. Gregory
Duronio, Robert J.
author_sort Penke, Taylor J.R.
collection PubMed
description A defining feature of heterochromatin is methylation of Lys9 of histone H3 (H3K9me), a binding site for heterochromatin protein 1 (HP1). Although H3K9 methyltransferases and HP1 are necessary for proper heterochromatin structure, the specific contribution of H3K9 to heterochromatin function and animal development is unknown. Using our recently developed platform to engineer histone genes in Drosophila, we generated H3K9R mutant flies, separating the functions of H3K9 and nonhistone substrates of H3K9 methyltransferases. Nucleosome occupancy and HP1a binding at pericentromeric heterochromatin are markedly decreased in H3K9R mutants. Despite these changes in chromosome architecture, a small percentage of H3K9R mutants complete development. Consistent with this result, expression of most protein-coding genes, including those within heterochromatin, is similar between H3K9R and controls. In contrast, H3K9R mutants exhibit increased open chromatin and transcription from piRNA clusters and transposons, resulting in transposon mobilization. Hence, transposon silencing is a major developmental function of H3K9.
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spelling pubmed-50246842017-02-15 Direct interrogation of the role of H3K9 in metazoan heterochromatin function Penke, Taylor J.R. McKay, Daniel J. Strahl, Brian D. Matera, A. Gregory Duronio, Robert J. Genes Dev Research Paper A defining feature of heterochromatin is methylation of Lys9 of histone H3 (H3K9me), a binding site for heterochromatin protein 1 (HP1). Although H3K9 methyltransferases and HP1 are necessary for proper heterochromatin structure, the specific contribution of H3K9 to heterochromatin function and animal development is unknown. Using our recently developed platform to engineer histone genes in Drosophila, we generated H3K9R mutant flies, separating the functions of H3K9 and nonhistone substrates of H3K9 methyltransferases. Nucleosome occupancy and HP1a binding at pericentromeric heterochromatin are markedly decreased in H3K9R mutants. Despite these changes in chromosome architecture, a small percentage of H3K9R mutants complete development. Consistent with this result, expression of most protein-coding genes, including those within heterochromatin, is similar between H3K9R and controls. In contrast, H3K9R mutants exhibit increased open chromatin and transcription from piRNA clusters and transposons, resulting in transposon mobilization. Hence, transposon silencing is a major developmental function of H3K9. Cold Spring Harbor Laboratory Press 2016-08-15 /pmc/articles/PMC5024684/ /pubmed/27566777 http://dx.doi.org/10.1101/gad.286278.116 Text en © 2016 Penke et al.; Published by Cold Spring Harbor Laboratory Press http://creativecommons.org/licenses/by-nc/4.0/ This article is distributed exclusively by Cold Spring Harbor Laboratory Press for the first six months after the full-issue publication date (see http://genesdev.cshlp.org/site/misc/terms.xhtml). After six months, it is available under a Creative Commons License (Attribution-NonCommercial 4.0 International), as described at http://creativecommons.org/licenses/by-nc/4.0/.
spellingShingle Research Paper
Penke, Taylor J.R.
McKay, Daniel J.
Strahl, Brian D.
Matera, A. Gregory
Duronio, Robert J.
Direct interrogation of the role of H3K9 in metazoan heterochromatin function
title Direct interrogation of the role of H3K9 in metazoan heterochromatin function
title_full Direct interrogation of the role of H3K9 in metazoan heterochromatin function
title_fullStr Direct interrogation of the role of H3K9 in metazoan heterochromatin function
title_full_unstemmed Direct interrogation of the role of H3K9 in metazoan heterochromatin function
title_short Direct interrogation of the role of H3K9 in metazoan heterochromatin function
title_sort direct interrogation of the role of h3k9 in metazoan heterochromatin function
topic Research Paper
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5024684/
https://www.ncbi.nlm.nih.gov/pubmed/27566777
http://dx.doi.org/10.1101/gad.286278.116
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