HIV-1 Tat-shortened neurite outgrowth through regulation of microRNA-132 and its target gene expression

BACKGROUND: Synaptodendritic damage is a pathological hallmark of HIV-associated neurocognitive disorders, and HIV-1 Tat protein is known to cause such injury in the central nervous system. In this study, we aimed to determine the molecular mechanisms of Tat-induced neurite shortening, specifically...

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Detalles Bibliográficos
Autores principales: Rahimian, Pejman, He, Johnny J.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2016
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5025601/
https://www.ncbi.nlm.nih.gov/pubmed/27634380
http://dx.doi.org/10.1186/s12974-016-0716-2

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5025601/
https://www.ncbi.nlm.nih.gov/pubmed/27634380
http://dx.doi.org/10.1186/s12974-016-0716-2

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