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HIV-1 Tat-shortened neurite outgrowth through regulation of microRNA-132 and its target gene expression
BACKGROUND: Synaptodendritic damage is a pathological hallmark of HIV-associated neurocognitive disorders, and HIV-1 Tat protein is known to cause such injury in the central nervous system. In this study, we aimed to determine the molecular mechanisms of Tat-induced neurite shortening, specifically...
| Autores principales: | , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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BioMed Central
2016
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5025601/ https://www.ncbi.nlm.nih.gov/pubmed/27634380 http://dx.doi.org/10.1186/s12974-016-0716-2 |