Cargando…
The CORM ALF-186 Mediates Anti-Apoptotic Signaling via an Activation of the p38 MAPK after Ischemia and Reperfusion Injury in Retinal Ganglion Cells
PURPOSE: Ischemia and reperfusion injury may induce apoptosis and lead to sustained tissue damage and loss of function, especially in neuronal organs. While carbon monoxide is known to exert protective effects after various harmful events, the mechanism of carbon monoxide releasing molecules in neur...
Autores principales: | Ulbrich, Felix, Kaufmann, Kai B., Meske, Alexander, Lagrèze, Wolf A., Augustynik, Michael, Buerkle, Hartmut, Ramao, Carlos C., Biermann, Julia, Goebel, Ulrich |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2016
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5072679/ https://www.ncbi.nlm.nih.gov/pubmed/27764224 http://dx.doi.org/10.1371/journal.pone.0165182 |
Ejemplares similares
-
The Carbon monoxide releasing molecule ALF-186 mediates anti-inflammatory and neuroprotective effects via the soluble guanylate cyclase ß1 in rats’ retinal ganglion cells after ischemia and reperfusion injury
por: Ulbrich, Felix, et al.
Publicado: (2017) -
Neuroprotection and neuroregeneration of retinal ganglion cells after intravitreal carbon monoxide release
por: Stifter, Julia, et al.
Publicado: (2017) -
Argon Mediates Anti-Apoptotic Signaling and Neuroprotection via Inhibition of Toll-Like Receptor 2 and 4
por: Ulbrich, Felix, et al.
Publicado: (2015) -
Hydrogen Sulfide Reduces Ischemia and Reperfusion Injury in Neuronal Cells in a Dose- and Time-Dependent Manner
por: Scheid, Stefanie, et al.
Publicado: (2021) -
Inhalative as well as Intravenous Administration of H(2)S Provides Neuroprotection after Ischemia and Reperfusion Injury in the Rats’ Retina
por: Scheid, Stefanie, et al.
Publicado: (2022)