Loss of endogenous thymosin β(4) accelerates glomerular disease

Glomerular disease is characterized by morphologic changes in podocyte cells accompanied by inflammation and fibrosis. Thymosin β(4) regulates cell morphology, inflammation, and fibrosis in several organs and administration of exogenous thymosin β(4) improves animal models of unilateral ureteral obs...

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Detalles Bibliográficos
Autores principales: Vasilopoulou, Elisavet, Kolatsi-Joannou, Maria, Lindenmeyer, Maja T., White, Kathryn E., Robson, Michael G., Cohen, Clemens D., Sebire, Neil J., Riley, Paul R., Winyard, Paul J., Long, David A.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2016
Materias:
Basic Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5073078/
https://www.ncbi.nlm.nih.gov/pubmed/27575556
http://dx.doi.org/10.1016/j.kint.2016.06.032

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5073078/
https://www.ncbi.nlm.nih.gov/pubmed/27575556
http://dx.doi.org/10.1016/j.kint.2016.06.032

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