Exogenous carbon monoxide inhibits neutrophil infiltration in LPS-induced sepsis by interfering with FPR1 via p38 MAPK but not GRK2

Excessive neutrophil infiltration in vital organs is life-threatening to patients who suffer from sepsis. We identified a critical role of exogenous carbon monoxide (CO) in the inhibition of neutrophil infiltration during lipopolysaccharide (LPS)-induced sepsis. CO delivered from carbon monoxide-rel...

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Detalles Bibliográficos
Autores principales: Wang, Xu, Qin, Weiting, Song, Mingming, Zhang, Yisen, Sun, Bingwei
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Impact Journals LLC 2016
Materias:
Research Paper
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5085153/
https://www.ncbi.nlm.nih.gov/pubmed/27144520
http://dx.doi.org/10.18632/oncotarget.9084

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5085153/
https://www.ncbi.nlm.nih.gov/pubmed/27144520
http://dx.doi.org/10.18632/oncotarget.9084

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