Procyanidin A2 Modulates IL-4-Induced CCL26 Production in Human Alveolar Epithelial Cells

Allergic asthma is an inflammatory lung disease that is partly sustained by the chemokine eotaxin-3 (CCL26), which extends eosinophil migration into tissues long after allergen exposure. Modulation of CCL26 could represent a means to mitigate airway inflammation. Here we evaluated procyanidin A2 as...

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Detalles Bibliográficos
Autores principales: Coleman, Sara L., Kruger, Marlena C., Sawyer, Gregory M., Hurst, Roger D.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2016
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5133887/
https://www.ncbi.nlm.nih.gov/pubmed/27845745
http://dx.doi.org/10.3390/ijms17111888

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5133887/
https://www.ncbi.nlm.nih.gov/pubmed/27845745
http://dx.doi.org/10.3390/ijms17111888

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