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Procyanidin A2 Modulates IL-4-Induced CCL26 Production in Human Alveolar Epithelial Cells

Allergic asthma is an inflammatory lung disease that is partly sustained by the chemokine eotaxin-3 (CCL26), which extends eosinophil migration into tissues long after allergen exposure. Modulation of CCL26 could represent a means to mitigate airway inflammation. Here we evaluated procyanidin A2 as...

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Detalles Bibliográficos
Autores principales:	Coleman, Sara L., Kruger, Marlena C., Sawyer, Gregory M., Hurst, Roger D.
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	MDPI 2016
Materias:	Article
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5133887/ https://www.ncbi.nlm.nih.gov/pubmed/27845745 http://dx.doi.org/10.3390/ijms17111888

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