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β-Amyloid triggers aberrant over-scaling of homeostatic synaptic plasticity
The over-production of β-amyloid (Aβ) has been strongly correlated to neuronal dysfunction and altered synaptic plasticity in Alzheimer’s disease (AD). Accordingly, it has been proposed that disrupted synaptic transmission and neuronal network instability underlie memory failure that is evident in t...
Autores principales: | , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2016
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5154098/ https://www.ncbi.nlm.nih.gov/pubmed/27955702 http://dx.doi.org/10.1186/s40478-016-0398-0 |