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Targeting neuronal MAPK14/p38α activity to modulate autophagy in the Alzheimer disease brain
Dysregulated autophagic-lysosomal degradation of proteins has been linked to the most common genetic defect in familial Alzheimer disease, and has been correlated with disease progression in both human disease and in animal models. Recently, it was demonstrated that the expression of MAPK14/p38α pro...
| Autores principales: | , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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Taylor & Francis
2016
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5173254/ https://www.ncbi.nlm.nih.gov/pubmed/27715387 http://dx.doi.org/10.1080/15548627.2016.1238555 |