Heme oxygenase-2 gene deletion attenuates oxidative stress in neurons exposed to extracellular hemin

BACKGROUND: Hemin, the oxidized form of heme, accumulates in intracranial hematomas and is a potent oxidant. Growing evidence suggests that it contributes to delayed injury to surrounding tissue, and that this process is affected by the heme oxygenase enzymes. In a prior study, heme oxygenase-2 gene...

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Detalles Bibliográficos
Autores principales: Regan, Raymond F, Chen, Jing, Benvenisti-Zarom, Luna
Formato: Texto
Lenguaje:English
Publicado: BioMed Central 2004
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC521072/
https://www.ncbi.nlm.nih.gov/pubmed/15377391
http://dx.doi.org/10.1186/1471-2202-5-34

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC521072/
https://www.ncbi.nlm.nih.gov/pubmed/15377391
http://dx.doi.org/10.1186/1471-2202-5-34

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