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Heme oxygenase-2 gene deletion attenuates oxidative stress in neurons exposed to extracellular hemin
BACKGROUND: Hemin, the oxidized form of heme, accumulates in intracranial hematomas and is a potent oxidant. Growing evidence suggests that it contributes to delayed injury to surrounding tissue, and that this process is affected by the heme oxygenase enzymes. In a prior study, heme oxygenase-2 gene...
| Autores principales: | , , |
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| Formato: | Texto |
| Lenguaje: | English |
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BioMed Central
2004
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC521072/ https://www.ncbi.nlm.nih.gov/pubmed/15377391 http://dx.doi.org/10.1186/1471-2202-5-34 |