Copper Enhances Zinc-Induced Neurotoxicity and the Endoplasmic Reticulum Stress Response in a Neuronal Model of Vascular Dementia

Zinc (Zn), an essential trace element, is secreted by synaptic vesicles during neuronal excitation and plays several critical roles in neuronal information processing. However, excess Zn ion (Zn(2+)) is neurotoxic and has a causative role in the pathogenesis of vascular dementia. Here, we investigat...

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Detalles Bibliográficos
Autores principales: Tanaka, Ken-ichiro, Kawahara, Masahiro
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2017
Materias:
Neuroscience
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5299027/
https://www.ncbi.nlm.nih.gov/pubmed/28232787
http://dx.doi.org/10.3389/fnins.2017.00058

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5299027/
https://www.ncbi.nlm.nih.gov/pubmed/28232787
http://dx.doi.org/10.3389/fnins.2017.00058

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