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Copper Enhances Zinc-Induced Neurotoxicity and the Endoplasmic Reticulum Stress Response in a Neuronal Model of Vascular Dementia
Zinc (Zn), an essential trace element, is secreted by synaptic vesicles during neuronal excitation and plays several critical roles in neuronal information processing. However, excess Zn ion (Zn(2+)) is neurotoxic and has a causative role in the pathogenesis of vascular dementia. Here, we investigat...
| Autores principales: | , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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Frontiers Media S.A.
2017
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5299027/ https://www.ncbi.nlm.nih.gov/pubmed/28232787 http://dx.doi.org/10.3389/fnins.2017.00058 |