Ginsenoside Rb1 Inhibits Doxorubicin-Triggered H9C2 Cell Apoptosis via Aryl Hydrocarbon Receptor

Doxorubicin (DOX) is a highly effective chemotherapeutic agent; however, the dose-dependent cardiotoxicity associated with DOX significantly limits its clinical application. In the present study, we investigated whether Rb1 could prevent DOX-induced apoptosis in H9C2 cells via aryl hydrocarbon recep...

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Detalles Bibliográficos
Autores principales: Zhang, Yaxin, Wang, Yuguang, Ma, Zengchun, Liang, Qiande, Tang, Xianglin, Tan, Hongling, Xiao, Chengrong, Gao, Yue
Formato: Online Artículo Texto
Lenguaje:English
Publicado: The Korean Society of Applied Pharmacology 2017
Materias:
Original Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5340546/
https://www.ncbi.nlm.nih.gov/pubmed/27829271
http://dx.doi.org/10.4062/biomolther.2016.066

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5340546/
https://www.ncbi.nlm.nih.gov/pubmed/27829271
http://dx.doi.org/10.4062/biomolther.2016.066

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