Cargando…
Chronic Ca(2+) influx through voltage-dependent Ca(2+) channels enhance delayed rectifier K(+) currents via activating Src family tyrosine kinase in rat hippocampal neurons
Excessive influx and the subsequent rapid cytosolic elevation of Ca(2+) in neurons is the major cause to induce hyperexcitability and irreversible cell damage although it is an essential ion for cellular signalings. Therefore, most neurons exhibit several cellular mechanisms to homeostatically regul...
Autores principales: | Yang, Yoon-Sil, Jeon, Sang-Chan, Kim, Dong-Kwan, Eun, Su-Yong, Jung, Sung-Cherl |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
The Korean Physiological Society and The Korean Society of Pharmacology
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5343060/ https://www.ncbi.nlm.nih.gov/pubmed/28280420 http://dx.doi.org/10.4196/kjpp.2017.21.2.259 |
Ejemplares similares
-
Neuronal input triggers Ca(2+) influx through AMPA receptors and voltage‐gated Ca(2+) channels in oligodendrocytes
por: Barron, Tara, et al.
Publicado: (2019) -
Ca(2+) Influx in T Cells: How Many Ca(2+) Channels?
por: Feske, Stefan
Publicado: (2013) -
Potentiated L-type Ca(2+) Channels Rectify
por: Leuranguer, Valérie, et al.
Publicado: (2003) -
STIMulating Ca(2+) influx
por: LeBrasseur, Nicole
Publicado: (2006) -
Impaired Fear Extinction Due to a Deficit in Ca(2+) Influx Through L-Type Voltage-Gated Ca(2+) Channels in Mice Deficient for Tenascin-C
por: Morellini, Fabio, et al.
Publicado: (2017)