The metabolic bone disease associated with the Hyp mutation is independent of osteoblastic HIF1α expression

Fibroblast growth factor-23 (FGF23) controls key responses to systemic phosphate increases through its phosphaturic actions on the kidney. In addition to stimulation by phosphate, FGF23 positively responds to iron deficiency anemia and hypoxia in rodent models and in humans. The disorder X-linked hy...

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Detalles Bibliográficos
Autores principales: Hum, Julia M., Clinkenbeard, Erica L., Ip, Colin, Cass, Taryn A., Allen, Matt, White, Kenneth E.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2017
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5365303/
https://www.ncbi.nlm.nih.gov/pubmed/28377980
http://dx.doi.org/10.1016/j.bonr.2017.01.003

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5365303/
https://www.ncbi.nlm.nih.gov/pubmed/28377980
http://dx.doi.org/10.1016/j.bonr.2017.01.003

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