Inhibition of c-Jun NH(2)-terminal kinase or extracellular signal-regulated kinase improves lung injury

BACKGROUND: Although in vitro studies have determined that the activation of mitogen-activated protein (MAP) kinases is crucial to the activation of transcription factors and regulation of the production of proinflammatory mediators, the roles of c-Jun NH(2)-terminal kinase (JNK) and extracellular s...

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Detalles Bibliográficos
Autores principales: Lee, Hui Su, Kim, Hee Jae, Moon, Chang Sook, Chong, Young Hae, Kang, Jihee Lee
Formato: Texto
Lenguaje:English
Publicado: BioMed Central 2004
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC538282/
https://www.ncbi.nlm.nih.gov/pubmed/15566575
http://dx.doi.org/10.1186/1465-9921-5-23

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC538282/
https://www.ncbi.nlm.nih.gov/pubmed/15566575
http://dx.doi.org/10.1186/1465-9921-5-23

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