Cargando…
KChIP2 regulates the cardiac Ca(2+) transient and myocyte contractility by targeting ryanodine receptor activity
Pathologic electrical remodeling and attenuated cardiac contractility are featured characteristics of heart failure. Coinciding with these remodeling events is a loss of the K(+) channel interacting protein, KChIP2. While, KChIP2 enhances the expression and stability of the Kv4 family of potassium c...
Autores principales: | Nassal, Drew M., Wan, Xiaoping, Liu, Haiyan, Laurita, Kenneth R., Deschênes, Isabelle |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5383259/ https://www.ncbi.nlm.nih.gov/pubmed/28384221 http://dx.doi.org/10.1371/journal.pone.0175221 |
Ejemplares similares
-
KChIP2 is a core transcriptional regulator of cardiac excitability
por: Nassal, Drew M, et al.
Publicado: (2017) -
Myocardial KChIP2 Expression in Guinea Pig Resolves an Expanded Electrophysiologic Role
por: Nassal, Drew M., et al.
Publicado: (2016) -
Specific effects of KChIP3/calsenilin/DREAM, but not KChIPs 1, 2 and 4, on calcium signalling and regulated secretion in PC12 cells
por: Venn, Neil, et al.
Publicado: (2008) -
Impact of KChIP2 on Cardiac Electrophysiology and the Progression of Heart Failure
por: Grubb, Søren, et al.
Publicado: (2012) -
NHE Isoform Switching and KChIP2 Upregulation in Aging Porcine Atria
por: Kant, Ritu, et al.
Publicado: (2013)