RelA/p65 inhibition prevents tendon adhesion by modulating inflammation, cell proliferation, and apoptosis

Peritendinous tissue fibrosis which leads to poor tendon function is a worldwide clinical problem; however, its mechanism remains unclear. Transcription factor RelA/p65, an important subunit in the NF-κB complex, is known to have a critical role in many fibrotic diseases. Here, we show that RelA/p65...

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Detalles Bibliográficos
Autores principales: Chen, Shuai, Jiang, Shichao, Zheng, Wei, Tu, Bing, Liu, Shen, Ruan, Hongjiang, Fan, Cunyi
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group 2017
Materias:
Original Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5386538/
https://www.ncbi.nlm.nih.gov/pubmed/28358376
http://dx.doi.org/10.1038/cddis.2017.135

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5386538/
https://www.ncbi.nlm.nih.gov/pubmed/28358376
http://dx.doi.org/10.1038/cddis.2017.135

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