Vitexin protects against hypoxic-ischemic injury via inhibiting Ca(2+)/Calmodulin-dependent protein kinase II and apoptosis signaling in the neonatal mouse brain

Neonatal hypoxic-ischemic is a major cause of death and disability in neonates. In this study, we suggest for the first time that pretreatment with vitexin may suppress a pro-apoptotic signaling pathway in hypoxic-ischemic neuronal injury in neonates by inhibition of the phosphorylation of Ca(2+)/Ca...

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Detalles Bibliográficos
Autores principales: Min, Jia-Wei, Kong, Wei-Lin, Han, Song, Bsoul, Nageeb, Liu, Wan-Hong, He, Xiao-Hua, Sanchez, Russell M., Peng, Bi-Wen
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Impact Journals LLC 2017
Materias:
Research Paper
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5421947/
https://www.ncbi.nlm.nih.gov/pubmed/28424420
http://dx.doi.org/10.18632/oncotarget.16065

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5421947/
https://www.ncbi.nlm.nih.gov/pubmed/28424420
http://dx.doi.org/10.18632/oncotarget.16065

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