Cargando…
Vitexin protects against hypoxic-ischemic injury via inhibiting Ca(2+)/Calmodulin-dependent protein kinase II and apoptosis signaling in the neonatal mouse brain
Neonatal hypoxic-ischemic is a major cause of death and disability in neonates. In this study, we suggest for the first time that pretreatment with vitexin may suppress a pro-apoptotic signaling pathway in hypoxic-ischemic neuronal injury in neonates by inhibition of the phosphorylation of Ca(2+)/Ca...
Autores principales: | Min, Jia-Wei, Kong, Wei-Lin, Han, Song, Bsoul, Nageeb, Liu, Wan-Hong, He, Xiao-Hua, Sanchez, Russell M., Peng, Bi-Wen |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Impact Journals LLC
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5421947/ https://www.ncbi.nlm.nih.gov/pubmed/28424420 http://dx.doi.org/10.18632/oncotarget.16065 |
Ejemplares similares
-
Vitexin reduces epilepsy after hypoxic ischemia in the neonatal brain via inhibition of NKCC1
por: Luo, Wen-di, et al.
Publicado: (2018) -
Ca(2+)/Calmodulin-Dependent Protein Kinase II Contributes to Hypoxic Ischemic Cell Death in Neonatal Hippocampal Slice Cultures
por: Lu, Qing, et al.
Publicado: (2013) -
Metabolic regulation of apoptosis via Ca+/Calmodulin Kinase II (CaMKII)
por: McCoy, Francis, et al.
Publicado: (2012) -
Resveratrol Inhibits Neuronal Apoptosis and Elevated Ca(2+)/Calmodulin-Dependent Protein Kinase II Activity in Diabetic Mouse Retina
por: Kim, Young-Hee, et al.
Publicado: (2010) -
Antinociceptive effects of vitexin in a mouse model of postoperative pain
por: Zhu, Qing, et al.
Publicado: (2016)