Bilirubin augments Ca(2+) load of developing bushy neurons by targeting specific subtype of voltage-gated calcium channels

Neonatal brain is particularly vulnerable to pathological levels of bilirubin which elevates and overloads intracellular Ca(2+), leading to neurotoxicity. However, how voltage-gated calcium channels (VGCCs) are functionally involved in excess calcium influx remains unknown. By performing voltage-cla...

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Detalles Bibliográficos
Autores principales: Liang, Min, Yin, Xin-Lu, Shi, Hai-Bo, Li, Chun-Yan, Li, Xin-Yi, Song, Ning-Ying, Shi, Hao-Song, Zhao, Yi, Wang, Lu-Yang, Yin, Shan-Kai
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2017
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5427978/
https://www.ncbi.nlm.nih.gov/pubmed/28348377
http://dx.doi.org/10.1038/s41598-017-00275-9

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5427978/
https://www.ncbi.nlm.nih.gov/pubmed/28348377
http://dx.doi.org/10.1038/s41598-017-00275-9

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