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Gap junction remodelling in human heart failure is associated with increased interaction of connexin43 with ZO-1
AIMS: Remodelling of gap junctions, involving reduction of total gap junction quantity and down-regulation of connexin43 (Cx43), contributes to the arrhythmic substrate in congestive heart failure. However, little is known of the underlying mechanisms. Recent studies from in vitro systems suggest th...
| Autores principales: | , , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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Oxford University Press
2008
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5436744/ https://www.ncbi.nlm.nih.gov/pubmed/18056766 http://dx.doi.org/10.1093/cvr/cvm083 |