TSA protects H9c2 cells against thapsigargin-induced apoptosis related to endoplasmic reticulum stress-mediated mitochondrial injury

Endoplasmic reticulum stress (ERS) activates an adaptive unfolded protein response (UPR) that facilitates cellular repair, however, under prolonged ER stress, the UPR can ultimately trigger apoptosis thereby terminating damaged cells. Recently, TSA has shown protective effects on ERS and its mechani...

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Detalles Bibliográficos
Autores principales: Li, Zhiping, Liu, Yan, Dai, Xinlun, Zhou, Qiangqiang, Liu, Xueli, Li, Zeyu, Chen, Xia
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2017
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5447435/
https://www.ncbi.nlm.nih.gov/pubmed/28579897
http://dx.doi.org/10.1016/j.jsps.2017.04.029

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5447435/
https://www.ncbi.nlm.nih.gov/pubmed/28579897
http://dx.doi.org/10.1016/j.jsps.2017.04.029

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