Voluntary saccade inhibition deficits correlate with extended white-matter cortico-basal atrophy in Huntington's disease

The ability to inhibit automatic versus voluntary saccade commands in demanding situations can be impaired in neurodegenerative diseases such as Huntington's disease (HD). These deficits could result from disruptions in the interaction between basal ganglia and the saccade control system. To in...

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Detalles Bibliográficos
Autores principales: Vaca-Palomares, Israel, Coe, Brian C., Brien, Donald C., Munoz, Douglas P., Fernandez-Ruiz, Juan
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2017
Materias:
Regular Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5472191/
https://www.ncbi.nlm.nih.gov/pubmed/28649493
http://dx.doi.org/10.1016/j.nicl.2017.06.007

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5472191/
https://www.ncbi.nlm.nih.gov/pubmed/28649493
http://dx.doi.org/10.1016/j.nicl.2017.06.007

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