Caveolin‐1 deficiency induces premature senescence with mitochondrial dysfunction

Paradoxical observations have been made regarding the role of caveolin‐1 (Cav‐1) during cellular senescence. For example, caveolin‐1 deficiency prevents reactive oxygen species‐induced cellular senescence despite mitochondrial dysfunction, which leads to senescence. To resolve this paradox, we re‐ad...

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Detalles Bibliográficos
Autores principales: Yu, Dong‐Min, Jung, Seung Hee, An, Hyoung‐Tae, Lee, Sungsoo, Hong, Jin, Park, Jun Sub, Lee, Hyun, Lee, Hwayeon, Bahn, Myeong‐Suk, Lee, Hyung Chul, Han, Na‐Kyung, Ko, Jesang, Lee, Jae‐Seon, Ko, Young‐Gyu
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2017
Materias:
Original Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5506423/
https://www.ncbi.nlm.nih.gov/pubmed/28514055
http://dx.doi.org/10.1111/acel.12606

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5506423/
https://www.ncbi.nlm.nih.gov/pubmed/28514055
http://dx.doi.org/10.1111/acel.12606

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