Hyperoxia results in increased aerobic metabolism following acute brain injury

Acute brain injury is associated with depressed aerobic metabolism. Below a critical mitochondrial pO(2) cytochrome c oxidase, the terminal electron acceptor in the mitochondrial respiratory chain, fails to sustain oxidative phosphorylation. After acute brain injury, this ischaemic threshold might b...

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Detalles Bibliográficos
Autores principales: Ghosh, Arnab, Highton, David, Kolyva, Christina, Tachtsidis, Ilias, Elwell, Clare E, Smith, Martin
Formato: Online Artículo Texto
Lenguaje:English
Publicado: SAGE Publications 2016
Materias:
Original Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5536254/
https://www.ncbi.nlm.nih.gov/pubmed/27837190
http://dx.doi.org/10.1177/0271678X16679171

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5536254/
https://www.ncbi.nlm.nih.gov/pubmed/27837190
http://dx.doi.org/10.1177/0271678X16679171

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