The NADPH oxidase NOX2 mediates loss of parvalbumin interneurons in traumatic brain injury: human autoptic immunohistochemical evidence

Pharmacological interventions for traumatic brain injury (TBI) are limited. Together with parvalbumin (PV) loss, increased production of reactive oxygen species (ROS) by the NADPH oxidase NOX enzymes represents a key step in TBI. Here, we investigated the contribution of NOX2-derived oxidative stres...

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Detalles Bibliográficos
Autores principales: Schiavone, Stefania, Neri, Margherita, Trabace, Luigia, Turillazzi, Emanuela
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2017
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5562735/
https://www.ncbi.nlm.nih.gov/pubmed/28821783
http://dx.doi.org/10.1038/s41598-017-09202-4

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5562735/
https://www.ncbi.nlm.nih.gov/pubmed/28821783
http://dx.doi.org/10.1038/s41598-017-09202-4

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