Cargando…
Haplodeficiency of Ataxia Telangiectasia Mutated Accelerates Heart Failure After Myocardial Infarction
BACKGROUND: Cell senescence is involved in the process of organ damage and repair; however, the underlying molecular mechanism needs to be further explored. METHODS AND RESULTS: Senescence‐related genes (ie, p21, p53, and ataxia telangiectasia mutated [ATM]) were shown to be elevated after myocardia...
Autores principales: | Jia, Lixin, Zhang, Wenmei, Ma, Youcai, Chen, Boya, Liu, Yan, Piao, Chunmei, Wang, Yuan, Yang, Min, Liu, Tingting, Zhang, Junmeng, Li, Taotao, Nie, Shaoping, Du, Jie |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5586323/ https://www.ncbi.nlm.nih.gov/pubmed/28724653 http://dx.doi.org/10.1161/JAHA.117.006349 |
Ejemplares similares
-
Senescent Cardiac Fibroblast Is Critical for Cardiac Fibrosis after Myocardial Infarction
por: Zhu, Fuli, et al.
Publicado: (2013) -
Ataxia-Telangiectasia Mutated Kinase: Role in Myocardial Remodeling
por: Thrasher, Patsy, et al.
Publicado: (2016) -
Deficiency of Ataxia Telangiectasia Mutated Kinase Delays Inflammatory Response in the Heart Following Myocardial Infarction
por: Daniel, Laura L., et al.
Publicado: (2014) -
Myoclonus in Ataxia–Telangiectasia
por: Termsarasab, Pichet, et al.
Publicado: (2015) -
Growth in ataxia telangiectasia
por: Natale, Valerie A. I., et al.
Publicado: (2021)