Methylglyoxal‐induced apoptosis is dependent on the suppression of c‐FLIP(L) expression via down‐regulation of p65 in endothelial cells

Methylglyoxal (MGO) is a reactive dicarbonyl metabolite of glucose, and its plasma levels are elevated in patients with diabetes. Studies have shown that MGO combines with the amino and sulphhydryl groups of proteins to form stable advanced glycation end products (AGEs), which are associated with va...

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Detalles Bibliográficos
Autores principales: Jang, Ji Hoon, Kim, Eun‐Ae, Park, Hye‐Jin, Sung, Eon‐Gi, Song, In‐Hwan, Kim, Joo‐Young, Woo, Chang‐Hoon, Doh, Kyung‐Oh, Kim, Kook Hyun, Lee, Tae‐Jin
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2017
Materias:
Original Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5661116/
https://www.ncbi.nlm.nih.gov/pubmed/28444875
http://dx.doi.org/10.1111/jcmm.13188

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5661116/
https://www.ncbi.nlm.nih.gov/pubmed/28444875
http://dx.doi.org/10.1111/jcmm.13188

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