Bax and caspases regulate increased production of mitochondria-derived reactive species in neuronal apoptosis: LACK of A role for depletion of cytochrome c from the mitochondrial electron transport chain

A Bax-dependent increase of reactive oxygen species (ROS) and other reactive species (RS) occurs after withdrawing NGF from mouse sympathetic neurons in cell culture. Possible mechanisms underlying the increased ROS/RS are leakage of electrons from the mitochondrial electron transport chain secondar...

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Detalles Bibliográficos
Autores principales: Kirkland, Rebecca A., Franklin, James L.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2015
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5668918/
https://www.ncbi.nlm.nih.gov/pubmed/29124200
http://dx.doi.org/10.1016/j.bbrep.2015.09.004

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5668918/
https://www.ncbi.nlm.nih.gov/pubmed/29124200
http://dx.doi.org/10.1016/j.bbrep.2015.09.004

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