2, 3, 7, 8‐Tetrachlorodibenzo‐p‐dioxin promotes endothelial cell apoptosis through activation of EP3/p38MAPK/Bcl‐2 pathway

Endothelial injury or dysfunction is an early event in the pathogenesis of atherosclerosis. Epidemiological and animal studies have shown that 2, 3, 7, 8‐tetrachlorodibenzo‐p‐dioxin (TCDD) exposure increases morbidity and mortality from chronic cardiovascular diseases, including atherosclerosis. How...

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Detalles Bibliográficos
Autores principales: Yu, Yu, Liu, Qian, Guo, Shumin, Zhang, Qianqian, Tang, Juan, Liu, Guizhu, Kong, Deping, Li, Juanjuan, Yan, Shuai, Wang, Ruiguo, Wang, Peilong, Su, Xiaoou, Yu, Ying
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2017
Materias:
Original Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5706494/
https://www.ncbi.nlm.nih.gov/pubmed/28699682
http://dx.doi.org/10.1111/jcmm.13265

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5706494/
https://www.ncbi.nlm.nih.gov/pubmed/28699682
http://dx.doi.org/10.1111/jcmm.13265

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