Cargando…
Dl-3n-butylphthalide reduces epileptiform activity through GluA2-lacking calcium-permeable AMPARs in epilepsy models
Epilepsy is the most prevalent chronic neurological disorder, and its pathological mechanism indicates that an imbalance between excitatory and inhibitory neurotransmission leads to neuronal hyperexcitability. Previous studies have suggested that dl-3n-butylphthalide (NBP) regulates the excitatory n...
Autores principales: | Yang, Qin, Hu, Yi-Da, Wang, Xue-Feng, Zheng, Fang-Shuo |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Impact Journals LLC
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5716726/ https://www.ncbi.nlm.nih.gov/pubmed/29228686 http://dx.doi.org/10.18632/oncotarget.21529 |
Ejemplares similares
-
Expression and Interaction Proteomics of GluA1- and GluA3-Subunit-Containing AMPARs Reveal Distinct Protein Composition
por: van der Spek, Sophie J. F., et al.
Publicado: (2022) -
GluA3 subunits are required for appropriate assembly of AMPAR GluA2 and GluA4 subunits on cochlear afferent synapses and for presynaptic ribbon modiolar–pillar morphology
por: Rutherford, Mark A, et al.
Publicado: (2023) -
Nitric Oxide Regulates GluA2-Lacking AMPAR Contribution to Synaptic Transmission of CA1 Apical but Not Basal Dendrites
por: Ivanova, Violetta O., et al.
Publicado: (2021) -
The GluA1 AMPAR subunit is necessary for hedonic responding but not hedonic value in female mice
por: Strickland, Jasmin A., et al.
Publicado: (2021) -
GluA1 AMPAR subunit deletion reduces the hedonic response to sucrose but leaves satiety and conditioned responses intact
por: Austen, Joseph M., et al.
Publicado: (2017)