CCL5-CCR5 interactions modulate metabolic events during tumor onset to promote tumorigenesis

BACKGROUND: In earlier studies we have shown that CCL5 activation of CCR5 induces the proliferation and survival of breast cancer cells in a mechanistic target of rapamycin (mTOR)-dependent manner and that this is in part due to CCR5-mediated increases in glycolytic metabolism. METHODS: Using the MD...

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Detalles Bibliográficos
Autores principales: Gao, Darrin, Cazares, Lisa H., Fish, Eleanor N.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2017
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5721608/
https://www.ncbi.nlm.nih.gov/pubmed/29216863
http://dx.doi.org/10.1186/s12885-017-3817-0

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5721608/
https://www.ncbi.nlm.nih.gov/pubmed/29216863
http://dx.doi.org/10.1186/s12885-017-3817-0

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