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CCL5-CCR5 interactions modulate metabolic events during tumor onset to promote tumorigenesis
BACKGROUND: In earlier studies we have shown that CCL5 activation of CCR5 induces the proliferation and survival of breast cancer cells in a mechanistic target of rapamycin (mTOR)-dependent manner and that this is in part due to CCR5-mediated increases in glycolytic metabolism. METHODS: Using the MD...
| Autores principales: | , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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BioMed Central
2017
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5721608/ https://www.ncbi.nlm.nih.gov/pubmed/29216863 http://dx.doi.org/10.1186/s12885-017-3817-0 |