Bile acid and cigarette smoke enhance the aggressive phenotype of esophageal adenocarcinoma cells by downregulation of the mitochondrial uncoupling protein-2

Limited information is available regarding mechanisms that link the known carcinogenic risk factors of gastro-esophageal reflux and cigarette smoking to metabolic alterations in esophageal adenocarcinoma (EAC). In the present study, we utilized a novel in-vitro model to examine whether bile acid and...

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Detalles Bibliográficos
Autores principales: Xu, Yuan, Feingold, Paul L., Surman, Deborah R., Brown, Kate, Xi, Sichuan, Davis, Jeremy L., Hernandez, Jonathan, Schrump, David S., Ripley, R. Taylor
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Impact Journals LLC 2017
Materias:
Research Paper
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5731855/
https://www.ncbi.nlm.nih.gov/pubmed/29254145
http://dx.doi.org/10.18632/oncotarget.22380

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5731855/
https://www.ncbi.nlm.nih.gov/pubmed/29254145
http://dx.doi.org/10.18632/oncotarget.22380

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