Cargando…
Palmitate-induced insulin resistance is attenuated by Pioglitazone and EGCG through reducing the gluconeogenic key enzymes expression in HepG2 cells
Hypothesis: Palmitate causes insulin resistance (IR) in insulin target tissue. Pioglitazone (an anti-hyperglycemic agent) and epigallocatechin gallate (EGCG, a dietary supplement) can be used for the treatment of type 2 diabetes. However, their molecular effects on gluconeogenesis remain unclear. Ob...
Autores principales: | Yadollah, S, Kazemipour, N, Bakhtiyari, S, Nazifi, S |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Carol Davila University Press
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5771254/ https://www.ncbi.nlm.nih.gov/pubmed/29362600 |
Ejemplares similares
-
Carnosic Acid and Carnosol Activate AMPK, Suppress Expressions of Gluconeogenic and Lipogenic Genes, and Inhibit Proliferation of HepG2 Cells
por: Hasei, Shun, et al.
Publicado: (2021) -
Synergy Analysis Reveals Association between Insulin Signaling and Desmoplakin Expression in Palmitate Treated HepG2 Cells
por: Wang, Xuewei, et al.
Publicado: (2011) -
Theaflavins Improve Insulin Sensitivity through Regulating Mitochondrial Biosynthesis in Palmitic Acid-Induced HepG2 Cells
por: Tong, Tuantuan, et al.
Publicado: (2018) -
Effect of Carotenoids from Phaeodactylum tricornutum on Palmitate-Treated HepG2 Cells
por: Mayer, Claire, et al.
Publicado: (2020) -
Involvement of Endoplasmic Reticulum Stress in Palmitate-induced Apoptosis in HepG2 Cells
por: Cho, Hyang-Ki, et al.
Publicado: (2008)